- Why pea is not shockable?
- Where does torsades de pointes originate?
- Is torsades a lethal rhythm?
- Do you have a pulse with torsades?
- What happens if you shock asystole?
- Which heart rhythms are lethal?
- Can you live a long life with long QT syndrome?
- Why does hypokalemia cause long QT?
- Does magnesium shorten QT interval?
- Can you shock Torsades de Pointes?
- What does torsades feel like?
- What are the 3 shockable rhythms?
- What is the most life threatening arrhythmia?
- Why is magnesium used for torsades?
- Is polymorphic v tach the same as torsades?
- What rhythms can you defibrillate?
- Which is the most lethal arrhythmia?
- What is the drug of choice for torsades de pointes?
- What can cause torsades?
Why pea is not shockable?
In PEA, there is electrical activity, but the heart either does not contract or there are other reasons this results in an insufficient cardiac output to generate a pulse and supply blood to the organs..
Where does torsades de pointes originate?
Conclusion: The most frequent site of origin of TdP is the outflow tract. Further studies are needed to understand why this relatively small area of the ventricle is a predominant site of origin of diverse ventricular arrhythmias.
Is torsades a lethal rhythm?
Most cases of torsades de pointes resolve on their own without treatment. However, it can develop into ventricular fibrillation, which can lead to cardiac arrest and may even be fatal.
Do you have a pulse with torsades?
Today one needs to be aware that drug-induced long QT syndrome is common and hence, a thorough medication history must be obtained. Patients with torsade may be hypotensive, have a rapid pulse and have loss of consciousness.
What happens if you shock asystole?
A single shock will cause nearly half of cases to revert to a more normal rhythm with restoration of circulation if given within a few minutes of onset. Pulseless electrical activity and asystole or flatlining (3 and 4), in contrast, are non-shockable, so they don’t respond to defibrillation.
Which heart rhythms are lethal?
Ventricular tachycardia (VT) and ventricular fibrillation (VF) are lethal cardiac arrhythmias, claiming a quarter million lives per year from sudden cardiac death (SCD).
Can you live a long life with long QT syndrome?
Living With Long QT syndrome (LQTS) usually is a lifelong condition. The risk of having an abnormal heart rhythm that leads to fainting or sudden cardiac arrest may lessen as you age. However, the risk never completely goes away.
Why does hypokalemia cause long QT?
Hypokalemia is another common risk factor in drug-induced LQTS. Low extracellular potassium paradoxically reduces IKr by enhanced inactivation  or exaggerated competitive block by sodium . As a result, hypokalemia prolongs the QT interval.
Does magnesium shorten QT interval?
Magnesium sulfate reduced the risk of an ibutilide- induced QTc interval increase of greater than 30 msec or greater than 60 msec and reduced the risk of a QTc interval value of more than 500 msec by 65%, 60%, and 68%, respectively (p=0.07, p=0.175, and p=0.160).
Can you shock Torsades de Pointes?
Key Points The long QT interval responsible for torsades de pointes ventricular tachycardia can be congenital or drug-induced. Immediate treatment of torsades is unsynchronized cardioversion beginning with 100 joules, although some patients respond to magnesium sulfate 2 g IV over 1 to 2 minutes.
What does torsades feel like?
You may suddenly feel your heart beating faster than normal, even when you’re at rest. In some TdP episodes, you may feel light-headed and faint. In the most serious cases, TdP can cause cardiac arrest or sudden cardiac death. It’s also possible have an episode (or more than one) that resolves quickly.
What are the 3 shockable rhythms?
Shockable Rhythms: Ventricular Tachycardia, Ventricular Fibrillation, Supraventricular Tachycardia.
What is the most life threatening arrhythmia?
Most sudden cardiac deaths are caused by abnormal heart rhythms called arrhythmias. The most common life-threatening arrhythmia is ventricular fibrillation, which is an erratic, disorganized firing of impulses from the ventricles (the heart’s lower chambers).
Why is magnesium used for torsades?
Magnesium is the drug of choice for suppressing early afterdepolarizations (EADs) and terminating the arrhythmia. Magnesium achieves this by decreasing the influx of calcium, thus lowering the amplitude of EADs. Magnesium can be given at 1-2 g IV initially in 30-60 seconds, which then can be repeated in 5-15 minutes.
Is polymorphic v tach the same as torsades?
Torsades de pointes (TdP) is a specific form of polymorphic ventricular tachycardia occurring in the context of QT prolongation; it has a characteristic morphology in which the QRS complexes “twist” around the isoelectric line. For TdP to be diagnosed, the patient has to have evidence of both PVT and QT prolongation.
What rhythms can you defibrillate?
Defibrillation – is the treatment for immediately life-threatening arrhythmias with which the patient does not have a pulse, ie ventricular fibrillation (VF) or pulseless ventricular tachycardia (VT). Cardioversion – is any process that aims to convert an arrhythmia back to sinus rhythm.
Which is the most lethal arrhythmia?
Arrhythmias that occur in the atria (the top chambers of the heart) are supraventricular (above the ventricles) in origin. These arrhythmias are not responsible for dramatic events such as sudden cardiac death, but the most common arrhythmia, atrial fibrillation, is supraventricular and can lead to fatal strokes.
What is the drug of choice for torsades de pointes?
Treatment of torsade de pointes includes: isoproterenol infusion, cardiac pacing, and intravenous atropine. Intravenous magnesium sulfate, a relatively new mode of therapy for torsade de pointes, was proven to be extremely effective and is now regarded as the treatment of choice for this arrhythmia.
What can cause torsades?
Risk factors for torsade include the following:Congenital long QT syndrome.Female gender.Acquired long QT syndrome (causes of which include medications and electrolyte disorders such as hypokalemia and hypomagnesemia)Bradycardia.Baseline electrocardiographic abnormalities.Renal or liver failure.